Revisión como estaba o 2 de setembro de 2018 ás 14:06 editar Miguelferig (conversa \| contribucións) 241.860 edicións →‎Uso terapéutico ← Edición máis vella		Revisión como estaba o 2 de setembro de 2018 ás 18:24 editar desfacer Miguelferig (conversa \| contribucións) 241.860 edicións →‎Factor Xa Edición máis nova →
Liña 68: [[Ficheiro:Coagulation in vivo-2.png\|miniatura\|350px\|Vías da coagulación do sangue in vivo mostrando o papel central da trombina.]] <!-- ~~Factor~~O factor Xa isé ~~the~~a ~~activated~~forma ~~form~~activada ~~of the~~do [[~~coagulation~~ factor de coagulación]] [[~~thrombokinase~~tromboquinase]], ~~known~~coñecido ~~eponymously~~polo asepónimo de factor de Stuart-Prower ~~factor~~. ~~Factor~~O factor X isé anun ~~[[enzyme]], a~~encima [[~~serine~~serina endopeptidase]], ~~which~~que ~~plays~~desempeña aun ~~key~~papel ~~role~~clave aten ~~several~~varias ~~stages~~enfermidades ~~of the~~co [[~~coagulation~~sistema de ~~system~~coagulación]]. Factor X is [[Biosynthesis\|synthesized]] in the [[liver]]. The most commonly used [[anticoagulants]] in clinical practice, [[warfarin]] and the [[heparin]] series of anticoagulants and [[fondaparinux]], act to inhibit the action of Factor Xa in various degrees. Traditional models of coagulation developed in the 1960s envisaged two separate cascades, the extrinsic [[tissue factor\|(tissue factor (TF))]] pathway and the intrinsic pathway. These pathways converge to a common point, the formation of the Factor Xa/Va complex which together with [[calcium]] and bound on a [[phospholipids]] surface generate [[thrombin\|thrombin (Factor IIa)]] from [[prothrombin\|prothrombin (Factor II)]].

Factor X: Diferenzas entre revisións